I am fascinated by the complex interactions that occur in the tears and at the ocular surface. The preocular fluid is organised by long, sugar-rich polymers, called mucins, whose physiology and biophysics I am investigating. Which are the structural characteristics that underpin the many postulated mucin roles at the ocular surface? How do disease-induced changes in, say, polymer length and in the number and nature of sugar chains that decorate it, how do these changes affect the quality of the preocular fluid? How do they affect interactions with bacteria? These are a few of the questions to which we are ...
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