5th
Brain-Immune Network Group Meeting
November 5th, 1999
The Jenner Centre, Bristol
Royal Infirmary, Lower Maudlin Street, Bristol, UK
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Scientific Programme
8:30 Registration
9:10 Chairman's Introduction
9:25 Vincent Geenen, University of Liege, Belgium
The immunological self of
neuroendocrine families: implications for self-tolerance and autoimmunity.
10:00 James Downing. University
of Cambridge, UK.
Involvement of
corticosteroids in the regulation of nitrergic cell abundance within the
thymus.
10:15 Thomas Connor. National
University of Ireland, Galway, Ireland
Fenfluramine provokes a
suppression of IL-1b and TNG-a secretion following
an in vivo challenge with lipipolysaccharide in rats.
10:30 Coffee
10:55 John Gruzelier. Imperial
College London, UK
Beneficial influences of
self-hypnosis training on immune and psychological functions.
11:30 Frank Hucklebridge.
University of Westminster, London, UK.
The influence of music
therapy in cancer patients upon salivary IgA and cortisol.
11:45 Sue Edwards. University of
Westminster, London, UK.
Analysis of components of the
diurnal pattern of salivary cortisol in relation to mood.
12:00 Posters
12:45 Lunch and Posterrs
continued
2:00 Dan Carr. University of Oklahoma, USA
Cytokine gene therapy in
herpes simplex virus.
2:35 Stephan von Hoersten. Hannover Medical School, Germany.
Postnatal maternal
deprivation and handling: effects on emotional behaviour in adult Lewis rats.
2:50 Torbjorn Breivik. University of Oslo, Norway
Postnatal maternal
deprivation and handling specifically alter peridontal disease susceptibility
in Lewis rats.
3:05 Coffee
3:30 Alberto Panerai. University of Milan, Italy
Does b-endorphin play a role in the modulation of Th1/Th2 responses?
4:05 Pierluigi Navarra. Catholic University School of Medicine, Rome,
Italy.
HIV gp120 protein acutely
stimulates CRF synthesis and release from the rat hypothalamus in vitro.
4:20 Ashley Grossman. Saint Bartholomew's Hospital, London, UK
Alterations of circulating
serum MIF levels in patiens with Cushing's disease and in normal subjects
following stimulation or suppression of the hypothalamo-pituitary-adrenal axis.
4:35 Chairman's Concluding Comments